We have finally discovered why COVID-19 causes heart attacks and strokes

 We have finally discovered why COVID-19 causes heart attacks and strokes

finally discovered

In the wake of the COVID-19 pandemic, a perplexing revelation has emerged - the virus's link to an increased risk of heart attacks and strokes. Recent research indicates that the SARS-CoV-2 virus directly infects the heart's arteries, exacerbating inflammation. This phenomenon could explain the heightened occurrence of strokes and heart attacks among affected patients.

The Puzzling Connection

Following an infection with SARS-CoV-2, the virus responsible for COVID-19, the risk of experiencing heart problems or a stroke is elevated for up to a year. While the virus's impact on the lungs and brain is relatively well-understood, its effects on the heart have remained somewhat enigmatic. A groundbreaking study delved into this issue, with the results published in Nature Cardiovascular Research.

Viral Presence in Atherosclerotic Plaques

The study's authors conducted their research using samples of arteries affected by atherosclerosis, taken from patients who had tragically succumbed to severe forms of the disease. Atherosclerosis is a condition characterized by the buildup of fatty plaques within the arteries, causing them to narrow. These plaques contain cholesterol-laden macrophages, cells typically tasked with removing cholesterol from the arteries. However, when overwhelmed, these macrophages become "fat" and promote plaque formation.

Remarkably, the authors revealed that the virus has an affinity for infecting these cholesterol-laden macrophages. This significantly escalates local inflammation. The macrophages, already struggling to eliminate cholesterol, fail to clear the virus and, instead, store it within the plaques.

When the Plaque Ruptures

The more inflamed a plaque becomes, the higher the risk of rupture. The presence of the virus within the plaque renders it exceedingly fragile. When a rupture occurs, plaque debris enters the bloodstream. This debris can form a clot that obstructs blood flow. If this transpires in the heart, it leads to a myocardial infarction, commonly known as a heart attack. If it happens in the brain, it results in a stroke. In other instances, the artery might fissure, causing another type of blockage.

Explaining Some Mysteries, But Not All

While this article sheds light on the consequences of a COVID-19 infection in patients with atherosclerosis, it does not provide answers to why some young and healthy individuals succumb to the disease. Moreover, the strains examined in this study are older and may not act the same as current variants. Nevertheless, these new findings emphasize the need for close monitoring of atherosclerosis patients who contract COVID-19.

In conclusion, this research underscores the multifaceted nature of COVID-19's impact on the human body. Beyond respiratory symptoms, it reveals how the virus can directly affect the heart, leading to potentially life-threatening complications.

FAQs

1. Can COVID-19 directly infect the heart?

Yes, recent research suggests that the SARS-CoV-2 virus can infect the arteries of the heart, increasing the risk of heart attacks and strokes.

2. How long does the heightened risk of heart problems and strokes last after a COVID-19 infection?

The increased risk can persist for up to a year after the initial infection.

3. What is atherosclerosis?

Atherosclerosis is a condition characterized by the accumulation of fatty plaques in the arteries, leading to narrowing and potential blockages.

4. Why do some young and healthy individuals die from COVID-19?

While this article provides insight into one aspect of COVID-19's impact, there are still mysteries surrounding the disease's varying effects on different individuals.

5. Should individuals with atherosclerosis be more cautious during the COVID-19 pandemic?

Yes, these findings emphasize the importance of closely monitoring atherosclerosis patients who contract COVID-19.